Xromi

Xromi

Generic Name

Xromi

Mechanism

Xromi competitively binds the adenosine triphosphate (ATP)‑binding cleft of the catalytic domains of JAK 1 and JAK 2, thereby preventing phosphorylation of signal‑transducer and activator of transcription (STAT) proteins.
• ↓ STAT nuclear translocation → ↓ pro‑inflammatory cytokine signaling (IL‑6, IFN‑γ, GM‑CSF)
• ↓ B‑cell proliferation and T‑cell activation
• ↓ immune‑mediated joint inflammation and cartilage degradation

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Pharmacokinetics

ParameterTypical ValueNotes
AbsorptionRapid; oral bioavailability ~40%Food reduces peak concentration (Cmin) by ~25%
DistributionVd ~ 50 LHighly protein‑bound (~85%) to albumin & α‑1‑acid glycoprotein
MetabolismPredominantly CYP3A4; minor CYP2D6Genotype‐linked variability (CYP3A4 poor metabolizers ↑ exposure)
Elimination90% via feces; 10% renalHalf‑life: 12–14 h (steady‑state ~5 days)
Drug interactionsStrong CYP3A4 or P‑gp inhibitors ↑ plasma levels (e.g., ketoconazole, clarithromycin); CYP3A4 inducers ↓ levels (e.g., rifampin)Dose adjustment or avoidance recommended

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Indications

  • Moderate‑to‑severe rheumatoid arthritis in adults with inadequate response or intolerance to at least one disease‑modifying anti‑rhythmic drug (DMARD)
  • *Off‑label* use reported for ankylosing spondylitis and psoriatic arthritis (case series only)

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Contraindications

Contraindication / WarningKey Points
Severe hepatic impairment (Child‑Pugh C)Contraindicated; avoid in cirrhosis
Active tuberculosis or other serious infectionsScreen prior to initiation; treat latent TB before starting
Uncontrolled malignancyCaution: potential oncogenic risk; monitor closely
Pregnancy / lactationCategory D; avoid; prefer alternative DMARDs
Thromboembolic events↑ risk of DVT/PE reported; avoid in patients with history of VTE
Cardiac disease (HF, arrhythmia)Monitor LVEF; cautious in heart failure patients
Cytopenias (ANC < 1.5 k/µL, platelets < 50 k/µL, Hb < 9 g/dL)Baseline CBC required; dose hold if thresholds breached

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Dosing

  • Initial dose: 2 mg orally once daily (QD)
  • Maintenance dose: 4 mg QD after achieving disease control (titration in 2 mg increments as tolerated)
  • Administration: Take with water, preferably at the same time each day; may be taken with food to improve tolerability but not required
  • Dose modification:
  • ↓ CYP3A4 inhibitors: consider 1 mg QD
  • ↑ CYP3A4 inhibitors: consider 1 mg every other day
  • Titration guidelines: Stop if CBC shows ANC < 1.0 k/µL or platelets < 25 k/µL; resume at 1 mg QD after recovery

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Adverse Effects

Adverse EffectFrequencyManagement
Upper respiratory tract infection~15%Symptomatic care
Nausea/vomiting~10%Take with food
Headache, dizziness~8%Bed rest, hydration
Anemia<5%CBC monitoring every 4 weeks
Neutropenia<4%CBC every 2 weeks initially
Thrombocytopenia<3%CBC monitoring
Herpes zoster3× ULN)**<1%Hold dose; re‑evaluate after resolution
Venous thromboembolism<1%Assess risk factors; consider prophylaxis
Hypertension<1%Monitor BP; antihypertensives as needed

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Monitoring

  • Baseline: CBC with differential, CMP (incl. LFTs), LVEF (echo/CMR), TB screening (IGRA or TST), pregnancy test (if applicable)
  • During therapy:
  • CBC & CMP every 2 weeks for first 3 months, then monthly
  • LFTs every 3 months thereafter
  • CBC annually thereafter if stable
  • LVEF at baseline, after 6 months, and as clinically indicated
  • Infections: Immediate evaluation for suspected infection; consider temporary hold
  • Pregnancy planning: Counsel patients of teratogenic risk; use effective contraception

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Clinical Pearls

  • Start low, go slow: Initiating at 2 mg avoids early cytopenias; many patients attain remission at 4 mg without dose escalation.
  • CYP3A4 co‑administration is the single biggest determinant of plasma levels: Use a drug interaction checker before adding antifungals, macrolides, or antiretrovirals.
  • Blood count drop‑watch: An isolated ANC fall of 20–25% from baseline can precede severe neutropenia; consider dose hold if ANC < 1.2 k/µL.
  • Herpes zoster prophylaxis: Vaccinate patients ≥50 years old before initiating *Xromi* to reduce zoster incidence by ~60%.
  • Cardiovascular vigilance: In patients with hypertension or heart failure, perform echocardiography at baseline and after 6 months; maintain ACEi/ARB therapy if feasible.
  • Thromboembolic risk stratification: In patients with a prior VTE or a history of VTE risk factors, a multidisciplinary review is advised before starting *Xromi*; consider low‑dose aspirin if appropriate.
  • Therapeutic drug monitoring is rarely needed: Because *Xromi*’s therapeutic window is wide, routine serum concentration monitoring is generally unnecessary; focus on clinical and laboratory surveillance instead.

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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