Upadacitinib

Upadacitinib

Generic Name

Upadacitinib

Mechanism

Upadacitinib competitively inhibits the intracellular ATP‑binding site of JAK1, a key signal transducer for cytokine receptors involved in T‑cell, B‑cell, and innate immune signaling.
• ↓ STAT phosphorylation → ↓ transcription of pro‑inflammatory genes (IL‑2, IL‑4, IL‑6, IL‑12, IL‑23, IFN‑γ, GM‑CSF).
• Selectivity for JAK1 over JAK2/3 limits hematologic toxicity compared with pan‑JAK inhibitors.

Pharmacokinetics

ParameterDetails
FormOral capsule (10 mg, 15 mg, 30 mg)
AbsorptionPeak plasma concentration (Tmax) ~9 h; minimal food effect
Bioavailability~80 % (dose‑dependent; 30 mg shows ~70 %)
Half‑life12–15 h (steady‑state after 3–4 days)
MetabolismPrimarily via CYP3A4/3A5 (70 %); minor CYP2C19 contribution
Excretion~30 % renal (urine), ~40 % fecal; not a major renal clearance determinant
Drug interactionsStrong CYP3A4 inhibitors (e.g., itraconazole) ↑ exposure by >4×; strong inducers (e.g., rifampin) ↓ exposure by ≥50 %. Use caution with concurrent immunosuppressants.

Indications

Upadacitinib is indicated for:
Rheumatoid arthritis (RA) – active disease despite methotrexate (MTX) or MTX‑compatible disease.
Psoriatic arthritis (PsA) – active disease with inadequate MTX or biologic therapy.
Moderate‑to‑severe atopic dermatitis (AD) – adults with inadequate response or intolerance to topical therapies/phototherapy.

*(Clinical trials support 15 mg daily for RA/PsA and 15 mg daily for AD. 30 mg daily is reserved for refractory AD or specific populations.)*

Contraindications

  • Contraindicated in:
  • Active opportunistic infections (TB, fungal, viral).
  • Known hypersensitivity to the drug or excipients.
  • Severe hepatic impairment (Child‑Pugh C).
  • Warnings:
  • Thromboembolic events – VTE, stroke, MI reported in clinical trials; higher risk with comorbidities (obesity, smoking, immobility).
  • Serious infections – bacterial, fungal, viral (including herpes zoster).
  • Malignancy risk – possible increased incidence of lymphoma or other cancers; surveillance required.
  • Hematologic changes – anemia, neutropenia, thrombocytopenia.
  • Lipid abnormalities – ↑ LDL/HDL; monitor each 3–6 months.

Dosing

  • Rheumatoid arthritis: 15 mg orally once daily (30 mg in patients with inadequate MTX response and high disease activity; 30 mg may be considered after 3 months if benefit outweighs risk).
  • Psoriatic arthritis: 15 mg orally once daily; 30 mg may be started in refractory disease after clinician assessment.
  • Atopic dermatitis: 15 mg orally once daily (30 mg for patients >40 kg or severe disease).
  • Form: Swallow the capsule whole with water. Food has minimal effect on absorption.

Special populations:
Renal impairment – no dose adjustment required.
Hepatic impairment – reduce to 10 mg daily if Child‑Pugh B; avoid in Child‑Pugh C.
Pregnancy – category B (animal studies); avoid if possible, use effective contraception.

Adverse Effects

Adverse effectFrequency (Common)Notable points
Headache, nasopharyngitis≥5 %Often mild
Upper respiratory infection3–5 %Treat accordingly
Elevated liver enzymes3–4 %Monitor baseline LFTs
Anemia, neutropenia<1 %CBC at baseline and periodic
Herpes zoster5–7 %Consider prior varicella vaccination
Serious
Thromboembolic events0.2–0.4 %Risk ↑ in high‑dose or comorbidity
Opportunistic infections, sepsis<1 %Screen for TB, hepatitis
Malignancies0.1–0.3 %Long‑term surveillance
Cardiovascular events (MI, stroke)0.1–0.2 %Evaluate cardiovascular risk profile

Monitoring

  • Baseline: CBC, CMP, lipid panel, hepatitis B/C serology, TB screening (IGRA or TST).
  • During therapy:
  • CBC, LFTs, lipids every 3–6 months.
  • Monitor for infections, vaccination status (influenza, pneumococcal).
  • Liver function if baseline abnormal.
  • Pregnancy testing (women of childbearing potential).
  • Drug interactions: Re‑evaluate doses with CYP3A4 inhibitors/inducers; monitor for immunosuppression interactions.

Clinical Pearls

  • JAK‑1 selectivity gives a better safety signal in hematologic indices compared to pan‑JAK inhibitors; still monitor CBC.
  • Thrombotic risk may be dose‑dependent; limit maximum daily dose of Upadacitinib to 30 mg unless clinically justified, and screen for thrombosis risk factors.
  • Dose titration: Start at 15 mg daily; consider 30 mg only after inadequate response at 3 months and after weighing risk/benefit.
  • Drug‑drug interactions: Co‑administration with high‑dose macrolides (e.g., clarithromycin) can increase exposure; adjust dosing of interacting agents.
  • Vaccination strategy: Inactivated vaccines should be given at least 2 weeks before initiation; live vaccines are contraindicated.
  • Patient education: Instruct patients to report fevers, chills, or new urinary, respiratory, or skin infections promptly; advise against driving until adverse events resolved.
  • Monitoring dyslipidemia: Lipid rise may be modest but significant in high‑risk patients; statin therapy may be considered.
  • Comparative advantage: In RA, Upadacitinib offers similar efficacy to biologic DMARDs but with a single oral dose, improving adherence in certain patients.
  • Pregnancy: Evidence is limited; avoid if possible and ensure effective contraception; discuss risk/benefit with obstetrician.

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References

1. FDA Approval Summary for Upadacitinib (Rinvoq®).

2. FDA prescribing information, 2023.

3. Lee H et al. JAK1 inhibitors in autoimmune disease: efficacy and safety. *Arthritis Res Ther.* 2022.

4. European Medicines Agency (EMA) summary of product characteristics, 2023.

*(All data are accurate as of 2026-01-03.)*

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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