Stribild

Stribild

Generic Name

Stribild

Mechanism

  • Elvitegravir: Binds the integrase catalytic core domain, inhibiting the insertion of viral DNA into the host genome.
  • Cobicistat: A potent inhibitor of CYP3A4, boosting elvitegravir exposure by reducing its metabolism.
  • Emtricitabine & Tenofovir disoproxil fumarate: Nucleoside reverse transcriptase inhibitors (NRTIs) that compete with natural nucleotides, causing chain termination during reverse transcription.

Together, the agents provide a tripartite blockade of HIV replication: reverse transcription (NRTIs), genome integration (integrase inhibitor), and pharmacokinetic amplification (cobicistat).

Pharmacokinetics

ComponentAbsorptionPeak Serum ConcentrationClearanceFood Effect
ElvitegravirRapid, ~1–3 h~2 h post‑dose71 % hepatic, 29 % renalSlight reduction without food
CobicistatRapid~0.5–2 hMainly hepaticNo significant effect
EmtricitabineRapid~1 hRenal (t½ 10 h)No effect
Tenofovir DFCumulative absorption; ~60 % of dose1.5–2 hRenal (t½ 17 h)No effect

Steady‑state achieved within 7 days.
Cobicistat provides >4‑fold elevation of elvitegravir AUC, permitting lower elvitegravir dosing.

Indications

  • First‑line therapy for treatment‑naïve adults and adolescents aged ≥10 years with HIV‑1.
  • Maintenance therapy for patients with no documented resistance to any component.
  • Barrier to resistance: Recommended only when viral genotypic testing confirms susceptibility to all four agents.

Contraindications

  • Severe renal impairment (CrCl < 40 mL/min) – Not recommended.
  • Leukopenia or neutropenia – Contraindicated.
  • Hypersensitivity to any constituent.
  • Concurrent strong CYP3A4 inducers (e.g., rifampin, carbamazepine) – contraindicated; may reduce elvitegravir levels.
  • Hepatotoxicity – Use cautiously; baseline LFTs required.
  • Pregnancy – No adequate data; use alternative regimen.
  • Tuberculosis co‑infection – Rifampin or rifabutin contraindicated.

Warnings
Renal dysfunction: May cause nephrotoxicity; monitor eGFR.
Bone demineralization: Tenofovir DF associated with decreased bone mineral density.
Metabolic effects: Minor lipid changes; monitor lipid panel in high‑risk patients.

Dosing

  • Dose: One tablet (elvitegravir 85 mg + cobicistat 150 mg + emtricitabine 200 mg + tenofovir DF 300 mg).
  • Frequency: Once daily, ±4 h of a regular meal.
  • Swallow whole; hydration encouraged.
  • Co‑administration: No special timing with other medications; avoid concurrent use of strong CYP3A inducers.

Missed Dose
• If ≤ 24 h past, take as soon as remembered.
• If > 24 h, skip and resume normal schedule; do not double dose.

Adverse Effects

CategoryCommon (≤ 10 %)Serious (≤ 1 %)
GINausea, vomiting, diarrhea, abdominal painSevere or persistent vomiting
CNSHeadache, dizziness, insomniaSevere headache, CNS depression
DermatologicRash, pruritusStevens‑Johnson syndrome
HematologicAnemia, leukopeniasevere neutropenia, aplastic anemia
RenalMild elevations in serum creatinineOliguric kidney injury, tubular necrosis
BoneLow‑grade bone demineralizationReduced bone mineral density, fractures
LipidHyperlipidemiaSevere hypertriglyceridemia
MetabolicMild hyperglycemiaLactic acidosis (rare)

Monitoring

ParameterFrequency
Viral Load (HIV‑RNA)Baseline, week 4, then every 12 weeks until suppressed.
CD4 CountBaseline, week 12, then every 24 weeks.
Serum Creatinine & eGFRBaseline, month 1, then quarterly.
Liver Enzymes (AST/ALT, bilirubin)Baseline, month 1, then quarterly.
Bone Mineral DensityBaseline if risk factors; repeat with DXA if clinical suspicion.
Lipid ProfileBaseline, month 3, then annually.
AdherenceVia pharmacy refill data, pill counts, or self‑report.

Other
• Monitor for drug interactions (e.g., antacids, antihypertensives, antiepileptics).
• If using cobicistat, assess CYP3A4/5 mediated metabolism of concomitant drugs.

Clinical Pearls

1. Cobicistat vs. Ritonavir – Cobicistat provides the same boosting effect with fewer gastrointestinal side effects, making it preferable for once‑daily fixed‑dose combinations.

2. Food is not mandatory but improves peak elvitegravir levels; countermassive meals can be used if patients experience nausea.

3. Renal CrCl ≥ 70 mL/min is optimal; in patients 40–70 mL/min, the FDA recommends dose appraisal as the benefits outweigh risks.

4. Use for Adolescents – Approved for patients ≥10 years; dose the same as adults but verify growth and development milestones.

5. Under-Resistance Testing – Because elvitegravir and NRTIs can rapidly select for resistant variants, genotypic resistance testing is mandatory before initiating if prior antiretroviral exposure exists.

6. Avoid with Strong Inducers – Rifampin, carbamazepine, and St. John’s wort markedly reduce elvitegravir concentrations; alternative H2 antagonists can be co‑prescribed safely.

7. Consider Bone Protection – In patients with risk factors for osteoporosis (post‑menopausal women, low BMI, smoking), evaluate for bone‑protective agents (vitamin D, calcium, bisphosphonates) concurrently.

8. Adherence is Key – The tri‑pill synergy offers a high genetic barrier; yet breakthrough viral replication often stems from non‑adherence, not drug resistance. Use electronic reminders or pill organizers.

9. Pregnancy Precautions – Tenofovir DF has restricted use in pregnancy; consider TDF (tenofovir disoproxil fumarate) formulations with known pregnancy safety data.

10. Drug–Drug Interactions – Prolonged proton pump inhibitor therapy may diminish cobicistat absorption slightly; schedule antacids ≥2 h before or after the dose.

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• *This drug card is optimized for quick reference by medical students, pharmacists, and clinicians seeking concise, evidence‑based information on Stribild within the context of HIV antiretroviral therapy.*

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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