Sodium bicarbonate

Sodium bicarbonate

Generic Name

Sodium bicarbonate

Mechanism

  • Bicarbonate ion (HCO₃⁻) acts as a buffer:
  • Reacts with excess hydrogen ions (H⁺) according to the equilibrium:

H⁺ + HCO₃⁻ ⇌ H₂CO₃ ⇌ CO₂ + H₂O
• This reaction consumes H⁺, raising the plasma pH and thereby correcting metabolic acidosis.
• In acid ingestion, it neutralizes free gastric acid, forming sodium chloride, water, and carbon dioxide, which is expelled by respiration.
• In dialysis settings, it buffers dialysate to maintain pH within 7.2–7.4, preventing diffusion of bicarbonate into the blood and reducing acid–base disturbances.

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Pharmacokinetics

ParameterKey Points
AbsorptionRapidly absorbed in the GI tract; 100 % bioavailability when taken orally.
DistributionDistributes primarily in the extracellular fluid; volume of distribution ≈ 0.45 L/kg.
MetabolismNo significant metabolic conversion.
EliminationRenal excretion via glomerular filtration and tubular secretion; half‑life ~5 h (shorter in renal disease).
Drug–Drug InteractionMay alter pH of plasma, affecting the ionization and clearance of other drugs (e.g., vancomycin, phenytoin).

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Indications

  • Acute or chronic metabolic acidosis (e.g., diabetic ketoacidosis, lactic acidosis, renal tubular acidosis).
  • Acute ingestion of acidic toxins (hydrofluoric acid, sulfuric acid).
  • Electrolyte correction (e.g., hypocalcemia secondary to acidosis).
  • Buffering of drug infusions (e.g., cytotoxic agents, antibiotics).
  • Dialysis – maintain dialysate pH; treat hyperkalemia in some protocols.
  • Emergency cardiac conditions – to mitigate hyperkalemia by shifting K⁺ intracellularly (short‑term, adjunctive).

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Contraindications

  • Severe respiratory failure / hypercapnia (CO₂ retention → paradoxical acidosis).
  • Advanced renal failure or acute kidney injury (impaired bicarbonate excretion).
  • Congestive heart failure/edema (risk of fluid overload).
  • Hypernatremia, hyponatremia, or severe electrolyte imbalance.
  • Pregnancy: Not teratogenic but use only if benefits outweigh risks.

Warnings:
• Monitor serum sodium, potassium, chloride, CO₂, and acid‑base status.
• Avoid rapid infusion rates to prevent hypernatremia, volume overload, and metabolic alkalosis.
• Use concomitant potassium supplementation when treating metabolic acidosis to prevent hypokalemia.

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Dosing

ScenarioTypical DoseRouteNotes
Metabolic acidosis (non‑renal)1–3 g (500–1500 mg) orally or IV per 100 mEq of deficitOral (pill/tablet) or IV (5 % NaHCO₃ 50 mEq/mL)Dose individualized by serum bicarbonate < 22 mEq/L and anion gap.
Metabolic acidosis (renal)1–3 g orally or 50–100 mEq (1–2 mEq/kg) IVOral or IVAdjust for renal impairment; monitor serum bicarbonate.
Acid ingestion1 mL/kg of 500 mEq/L solutionIV, but can give orally (if stable)First dose 500‑1000 mEq; repeat as needed; avoid hypotonic solutions.
Dialysis buffering3 M or 5 M (1–3 mL) in dialysateIV (in dialysis circuit)Adjust pH to 7.2–7.4.
Hyperkalemia2–3 mEq/kg IV (maximum 200 mEq)IVRapid infusion 4–5 mL/kg over 5–10 min; use with glucose–insulin.

Administration Tips:
• For IV use, add to 5–10 % dextrose (5 % D) to cushion osmolarity and avoid rapid osmotic shifts.
• Do not crush tablets; prefer liquid form for IV or urgent oral use.

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Adverse Effects

Adverse EffectCommonalitySeverityManagement
Nausea, vomiting, epigastric painCommonMildSlow or divide dose; consider antacids.
Hyponatremia / hypernatremiaPossibleModerate to severeFluid restriction/adjust dose; monitor electrolytes.
Metabolic alkalosisCommonModerateReassess dose, correct underlying cause, supplement electrolytes.
HypokalemiaCommonModerateSupplement potassium orally or IV.
Fluid overload & edemaPossibleModerateUse in cardiac patients cautiously; monitor weight/creatinine.
Hyperventilation / paradoxical acidosis (if CO₂ retention)RareSevereStop therapy, provide ventilatory support.
Severe hypersensitivity (rare)RareSevereDiscontinue; evaluate for underlying drug allergy.

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Monitoring

  • Arterial blood gases (ABG): pH, PaCO₂, PaO₂, bicarbonate level.
  • Serum electrolytes: Na⁺, K⁺, Cl⁻, HCO₃⁻, Ca²⁺.
  • Serum creatinine & BUN: renal function.
  • Urine output: renal perfusion.
  • Vital signs: BP, pulse, RR, SpO₂.
  • Fluid balance: input/output charts, daily weights.

Frequency:
Acute settings: every 30–60 min until stabilization.
Chronic/metabolic: daily or as clinically indicated.

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Clinical Pearls

1. Avoid in chronic respiratory acidosis – the bicarbonate load can worsen CO₂ retention; instead focus on improving ventilation.

2. Use 1.5 % solution when treating severe acidosis in dialysis – it mitigates rapid osmolar shifts and is easier to titrate to the target pH.

3. Potassium must accompany bicarbonate in metabolic acidosis – even if serum K⁺ is normal, bicarbonate will drive K⁺ into cells, precipitating dangerous hypokalemia.

4. Administer with caution in heart failure – the sodium load can precipitate pulmonary edema; consider a low‑sodium formulation or split dosing.

5. In toxic ingestions, give 500–1 g orally or IV initially, then reassess – rapid neutralization is key but avoid overshooting, as this can induce alkalosis.

6. Remember its dual role – not just an antacid but also an electrolyte buffer; monitor both acid–base and electrolyte panels.

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• *This drug card is for educational purposes and should be used in conjunction with institutional guidelines and the prescribing information provided by regulatory agencies.*

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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