Kenalog

Kenalog

Generic Name

Kenalog

Brand Names

for triamcinolone acetonide) is a potent synthetic glucocorticoid used for a variety of inflammatory, allergic, and autoimmune conditions. It is available in oral, topical, and injectable formulations and is favored for its high bioavailability and minimal mineralocorticoid activity.

Mechanism

  • Glucocorticoid receptor (GR) agonist: Triamcinolone enters cells via passive diffusion, binds the intracellular GR, and translocates to the nucleus.
  • Transrepression: Inhibits NF‑κB and AP‑1, reducing transcription of pro‑inflammatory cytokines (IL‑1, IL‑6, TNF‑α) and chemokines.
  • Transactivation: Stimulates expression of anti‑inflammatory proteins such as annexin‑1 and lipocortin‑1.
  • Immunomodulation: ↓ Lymphocyte proliferation, ↓ eosinophil recruitment, and ↓ mast cell degranulation.
  • Reduced vascular permeability and inhibition of bradykinin/serotonin pathways diminish edema and pain.

Pharmacokinetics

ParameterTypical ValueNotes
AbsorptionOral 50‑70 % bioavailability; intramuscular (IM) 100 %IM onset ~30 min; oral half‑life 3–4 h
DistributionExtensive; protein binding ~85 %Crosses placenta; breast‑milk minimal
MetabolismHepatic (CYP3A4, CYP1A2) → hydroxylated metabolitesNo active metabolites
EliminationRenal (≈40 %) and fecal (≈30 %)Half‑life 3–4 h; accumulation with repeated dosing
Special PopulationsRenal/hepatic impairment: dosage adjustments; pregnancy: category C

Indications

  • Ophthalmology: Allergic conjunctivitis, uveitis, posterior segment inflammation, post‑operative ocular inflammation.
  • Dermatology: Psoriasis, eczema, dermatitis, cutaneous sarcoidosis, alopecia areata.
  • Lung: Asthma exacerbations, allergic bronchopulmonary aspergillosis, chronic obstructive pulmonary disease (COPD) flare‑up.
  • Joint & Soft Tissue: Rheumatoid arthritis (intra‑articular), osteoarthritis, gout, tendinopathies, bursitis.
  • ENT: Allergic rhinitis, sinusitis, nasal polyposis; can be used intranasally.
  • Systemic: Autoimmune diseases (e.g., systemic lupus erythematosus) when oral therapy is indicated.

Contraindications

  • Contraindications: Active systemic fungal infections, untreated tuberculosis, systemic viral infections, hypersensitivity to triamcinolone or other corticosteroids.
  • Warnings:
  • Systemic exposure → adrenal insufficiency, Cushingoid changes, hypertension.
  • Ocular forms → ocular hypertension, glaucoma, cataract progression, secondary infection.
  • Topical use over large skin areas → systemic effects in children.
  • Use cautiously in diabetes, hypertension, osteoporosis, osteoporosis‑prone patients.

Dosing

IndicationFormTypical DoseFrequencyNotes
Intramuscular injectionKenalog 10 mg/2 mL10–40 mgEvery 4–6 weeksMonitor for local pain, muscle weakness
Topical cream/fabric0.1–0.5 % triamcinolone2 g once daily14–28 daysAvoid application >10 % body surface area
OphthalmicKenalog 0.1% (drops)4×/day5–14 daysUse only under ophthalmologist supervision
Intranasal1‑2 mg/day2–3 mg/day5–21 daysUse 1–2 sprays; alternate nostrils
Oral0.5‑2 mg/kg20–60 mg1–2 days for flareNot first‐line systemic therapy

> Dosing tip: For intra‑articular injections, combine Kenalog with local anesthetic (e.g., lidocaine) to reduce post‑injection pain.

Adverse Effects

Common (≤10 %)
• Mood changes, insomnia, increased appetite, hyperglycemia
• Local pain at injection site, muscle aches, skin thinning (topical)

Serious (>10 %)
• Systemic Cushing syndrome, adrenal suppression (especially with chronic use)
• Osteoporosis, osteonecrosis (particularly with high‑dose IM)
• Hypertension, hyperlipidemia, peptic ulcer disease
• Opportunistic infections (e.g., candidiasis, tuberculosis)
• Serious ocular complications (elevated intra‑ocular pressure, cataract formation)

Monitoring

ParameterFrequencyRationale
Serum glucoseBaseline, then 2–4 weeksPrevent diabetic decompensation
Blood pressureBaseline, then 2–4 weeksSteroid‑induced hypertension
Serum potassiumBaseline, then as indicatedRare hypokalemia with high‑dose steroids
Cortisol & ACTHBefore prolonged coursesDetect adrenal suppression
Bone density (DEXA)Baseline for >6 months useOsteoporosis prevention
Intra‑ocular pressureEvery 2 weeks in ocular useAvoid glaucoma risk
Skin assessmentBaseline, then monthly (topicals)Avoid atrophy, striae

Clinical Pearls

  • Kenalog vs. Hydrocortisone: Kenalog has ~5–10× greater potency and 13 % lower mineralocorticoid activity—ideal for conditions requiring strong anti‑inflammatory but minimal sodium retention.
  • Kenalog in Ophthalmology: The drug’s viscosity allows sustained release from ocular implants; a single intravitreal Kenalog 0.4 mg can provide up to 6 months of anti‑inflammatory effect.
  • Immunology: Despite high potency, Kenalog rarely induces systemic immunosuppression when used topically <10 % body area and for <4 weeks—this is why skin‑specific conditions often prefer it over systemic steroids.
  • Drug Interactions: Concomitant CYP3A4 inhibitors (ketoconazole, erythromycin) may elevate triamcinolone levels; conversely, rifampin can reduce efficacy.
  • Pediatric Use: Use lower doses than adult equivalents; monitor growth parameters, as chronic steroids can stunt growth velocity.
  • Withdrawal Strategy: For chronic IM therapy, taper slowly over 4–6 weeks with 10‑mg reductions per dose; oral prednisolone can be used as a bridging agent.
  • Pregnancy & Lactation: Kenalog is category C; use only if benefits outweigh risks, and avoid high‑dose systemic exposure. Low‑dose topical forms are generally considered safe if the area of contact is Final note: Kenalog remains a cornerstone in steroid therapy, offering high potency with a relatively favorable safety profile when used judiciously and monitored carefully.

Medical & AI Content Disclaimers
Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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