Intuniv

Intuniv

Generic Name

Intuniv

Mechanism

  • Selective α2A‑adrenergic agonist: preferentially activates presynaptic α2A receptors, reducing presynaptic norepinephrine release in the prefrontal cortex.
  • Enhances prefrontal cortical connectivity → improves working memory, impulse control, and executive function.
  • Lowered postsynaptic α2A activity mediates the neurophysiological effect, distinguishing it from non‑selective α2 agonists.

Pharmacokinetics

ParameterValue / Description
AbsorptionOral, delayed‑release capsules; peak plasma concentration (Tmax) ~6–8 h.
Bioavailability~8% after single‑dose oral administration (low due to first‑pass).
Half‑life~17–20 h (extended‑release formulation).
MetabolismHepatic via CYP2D6 and non‑enzymatic pathways; minimal involvement of CYP3A4/5.
ExcretionPrimarily renal (~25–30% unchanged), the rest as metabolites.
Steady‐stateAchieved after ~2–4 weeks of continuous dosing.

Indications

  • ADHD in patients 6–17 years (FDA‑approved).
  • Off‑label therapeutic use in adults with ADHD, agitation, or insomnia associated with ADHD.
  • May be combined with stimulants to enhance efficacy and reduce stimulant‑related side effects such as appetite suppression.

Contraindications

ContraindicationsWarnings
Hypersensitivity to guanfacine or excipients.Severe hypotension or postural orthostatic hypotension.
Bradycardia (HR < 55 bpm) or third‑degree AV block.Concomitant use of strong CYP2D6 inhibitors (e.g., fluoxetine) can elevate levels.
Significant hepatic dysfunction.Abrupt discontinuation may cause rebound hypertension; taper slowly.
Persistent sleepiness or excessive sedation—discontinue if severe.

Dosing

1. Initial dose: 1 mg once daily (usually in the evening).
2. Titration: Increase by 0.5–1 mg weekly as tolerated. Max recommended dose: 4 mg/day.
3. Administration: Chew or swallow the capsule whole; do not crush or chew (delayed‑release).
4. Timing: Take at the same time each day to maintain stable levels.
5. Food: Food does not significantly affect absorption; can be taken with or without meals.

*Example titration schedule for a 12‑year‑old:*

WeekDose (mg/day)
1–21
3–41.5
5–62.5
7+3.5–4.0 (if needed)

Adverse Effects

CategoryExamples
Common (≥5 %)Somnolence, fatigue, dry mouth, headache, dizziness, constipation, hypertension, initial hypotension.
Moderate (1–5 %)Bradycardia, decreased appetite, insomnia, insomnia, mild abdominal pain.
Serious (<1 %)Bradyarrhythmias (AV block), severe hypotension, syncope, marked sedation, anaphylaxis (rare).

Long‑term effects: Minimal growth suppression reported; monitor height/weight during routine follow‑up.

Monitoring

ParameterFrequency
Vital signs (BP, HR)Baseline, 1–2 weeks after dose change, then every 4–6 weeks.
Growth metrics (height, weight)Every visit or as per school health guidelines.
Serum creatinine / liver enzymesBaseline, then annually or if clinically indicated.
Sleep qualityAssess daily at titration, especially if somnolence reported.
Adverse eventsDocument at each visit; note any cardiovascular symptoms.

Clinical Pearls

  • Start low, titrate slow: The most common adverse reaction is hypotension; gradual dose escalation mitigates this risk.
  • Avoid sudden discontinuation: A rapid dose reduction can trigger rebound hypertension or tachycardia; taper by 0.5 mg per week.
  • Adjunct to stimulants: Adding Intuniv can reduce stimulant‑induced insomnia, appetite loss, and peak‑dose jitter.
  • Pediatric dosing precision: Use the liquid or chewable formulation for fine‑tuned dosing in younger children (under 6 kg).
  • Potential for improved sleep: Many patients note easier sleep onset; consider night‑time dosing for ADHD‑related insomnia.
  • Drug interactions: Concomitant use with CYP2D6 inhibitors (fluoxetine, paroxetine) may require closer BP monitoring.
  • Non‑cognitive benefits: Emerging evidence suggests Intuniv may slightly improve inhibitory control in children with comorbid oppositional defiant disorder.

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Intuniv remains a cornerstone for non‑stimulant ADHD management in pediatric populations, offering a mechanism distinct from stimulants, a favorable side‑effect profile when titrated appropriately, and synergistic benefits when combined with other ADHD therapies.

Medical & AI Content Disclaimers
Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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