Eylea

Eylea

Generic Name

Eylea

Mechanism

Eylea binds and neutralizes the following ligands:
VEGF‑A (all isoforms)
VEGF‑B
Placental growth factor (PlGF)

By sequestering these angiogenic mediators, the drug blocks activation of VEGF receptors on endothelial cells, inhibiting neovascularization, vascular leakage, and inflammation in the retina.

Pharmacokinetics

ParameterDetails
AdministrationIntravitreal injection (2 mg/0.05 mL)
DistributionPrimarily local to the vitreous; minimal systemic exposure
AbsorptionRapid intra‑vitreal distribution; peak concentration ≈1 day
Half‑lifeIntravitreal elimination half‑life ~9 days (≈4 weeks)
MetabolismProteolytic degradation into peptides; negligible hepatic metabolism
ExcretionVia the retinal and choroidal circulation; systemic levels cleared by renal & hepatic pathways (minimal systemic exposure)
Drug–Drug InteractionsNo clinically significant interactions; caution with systemic anti‑VEGF or anti‑platelet agents

Indications

  • Neovascular (wet) age‑related macular degeneration (AMD)
  • Diabetic macular edema (DME)
  • Macular edema secondary to central or branch retinal vein occlusion (CRVO/BRVO)
  • Myopic choroidal neovascularization
  • Other retinal diseases with VEGF‑mediated pathology (off‑label use)

Contraindications

  • Active ocular infection (e.g., endophthalmitis)
  • Severe ocular inflammation or uveitis
  • Known hypersensitivity to aflibercept or any component
  • Uncontrolled systemic hypertension (may exacerbate ocular hypertension)
  • Recent thromboembolic event (caution in patients with recent stroke or MI)

Warnings:
Endophthalmitis – rare but sight‑threatening; prompt recognition essential.
Intra‑ocular pressure elevation – monitor and manage appropriately.
Systemic cardiovascular events – rare; consider baseline cardiovascular assessment.

Dosing

  • Initial loading phase: 2 mg intravitreal injection every 4 weeks for 3 consecutive doses.
  • Maintenance phase: every 8 weeks (Q8W) based on disease activity.
  • Alternative regimens:
  • *Treat‑and‑Extend*: adjust interval by 2 weeks ± based on OCT and visual acuity.
  • *Pro‑Re‑Nata (PRN)*: treat as needed after initial loading.
  • Injection technique: sterile ocular prep, lid speculum, 30‑g needle, avoid reflux.

Reconstitution: Pre‑filled syringes (2 mg/0.05 mL) ready for use; no dilution required.

Adverse Effects

OcularSystemic
*Common*Intra‑ocular inflammation (hypopyon, anterior chamber cells)
Endophthalmitis (rare)
Increased intra‑ocular pressure
Vitreous hemorrhage
Cataract progression
SeriousSevere endophthalmitis
Acute rise in IOP leading to optic nerve damage
Macular edema progression
Systemic hypertension (rare)
Thromboembolic events (MI, stroke) – very uncommon

Monitoring

  • Baseline: Best‑corrected visual acuity (BCVA), optical coherence tomography (OCT), intra‑ocular pressure (IOP), slit‑lamp exam.
  • Follow‑up (every visit):
  • BCVA and OCT to assess edema/CRT.
  • IOP measurement; treat if ≥30 mmHg or >5 mmHg above baseline.
  • Ophthalmic examination for signs of inflammation or infection.
  • Long‑term: Document cumulative number of injections, visual outcomes, and any adverse events.

Clinical Pearls

  • Pearl 1 – Early “Loading” Matters: The 3‑dose 4‑week loading schedule dramatically reduces the risk of breakthrough macular edema compared to starting on an 8‑week interval.
  • Pearl 2 – Treat‑and‑Extend Yields Long‑Term Stability: Studies show that a treat‑and‑extend regimen maintains visual gains while reducing clinic visits and injections versus fixed Q8W.
  • Pearl 3 – Monitor IOP Even in “Inert” Eyes: A small subset (~3–5 %) of patients develop significant IOP elevation; baseline glaucoma history warrants pre‑emptive IOP control.
  • Pearl 4 – Systemic Safety Is Robust: Despite systemic VEGF inhibition, large‑scale trials report no excess cardiovascular events versus sham; still, consider cardiovascular history when initiating therapy.
  • Pearl 5 – Use of Anti‑Inflammatory Adjuncts: For patients with mild ocular inflammation, a short course of topical steroids post‑injection can blunt inflammatory reactions without compromising efficacy.

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• *This drug card consolidates current evidence and is designed for quick reference by medical students and practicing clinicians.*

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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