Yupelri

Yupelri

Generic Name

Yupelri

Mechanism

  • Selective TGF‑β superfamily ligand trap: Binds and neutralizes α‑chain ligands such as activin A, GDF‑11, and others that signal through the SMAD 2/3 pathway.
  • Enhances erythroid progenitor differentiation: By inhibiting SMAD‑dependent inhibition, it restores the maturation of late-stage erythroblasts, increasing endogenous erythropoiesis and reducing transfusion requirements.
  • Reduces ineffective erythropoiesis: Decreases erythrocyte destruction in the marrow and improves hemoglobin levels.

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Pharmacokinetics

ParameterValueClinical Notes
AbsorptionSubcutaneous (SC) administrationPeak serum concentrations within 3–5 days post‑dose.
DistributionProtein‑bound (~95 %); volume of distribution ≈ 300 mL/kgLimited ocular or CNS penetration.
MetabolismCleaved by proteases into active domain; no significant CYP involvementLow drug‑drug interaction potential via metabolic enzymes.
EliminationRenal and hepatobiliary clearance; half‑life ~13–16 daysDose adjustments not required for mild‑to‑moderate renal/hepatic impairment.
Loading Dose3 mg/kg SC on day 0, followed by maintenance every 3 weeksWeight‑based dosing optimizes therapeutic exposure.

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Indications

  • Transfusion‑dependent anemia in adults with MDS containing ring sideroblasts (RiMDS), not otherwise fit for or refractory to standard therapies.
  • Transfusion‑dependent β‑thalassemia (adult patients); reduces transfusion burden and improves hemoglobin maintenance.

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Contraindications

  • Contraindications
  • Hypersensitivity to any component of the formulation (e.g., recombinant proteins).
  • Active, uncontrolled infection at the injection site.
  • Warnings
  • Iron overload: Monitor iron status; concurrent chelation therapy may be necessary.
  • Cardiovascular events: Hypertension and thromboembolic events have been reported; monitor BP and assess cardiovascular history.
  • Bone marrow fibrosis: Rare reports of new‑onset fibrosis; monitor marrow adequacy.
  • Precautions
  • Pregnancy and lactation: Limited data; consider risk–benefit balance.
  • Elderly: Monitor with caution; no evidence of altered PK.
  • Comorbid cancers: Evaluate for potential impact on disease progression.

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Dosing

ConditionInitial DoseMaintenanceAdministration
RiMDS3 mg/kg SC (max 35 mg)Every 3 weeksSubcutaneous injection (anterolateral thigh or upper arm).
β‑thalassemia3 mg/kg SC (max 35 mg)Every 3 weeksSame as above.

SC Technique: Rotate injection sites, hold the needle for 30 s post‑injection to reduce intra‑vascular leakage.
Premedication: Not routinely required; antihistamines may be used if mild infusion reaction occurs.
Storage: Reconstituted product (1 mg/mL) stored at 2–8 °C; use within 24 h.

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Monitoring

ParameterFrequencyRationale
CBC (Hb, RBC, WBC, Platelets)At baseline, weekly for first 6 weeks, then every 3 weeksEvaluate efficacy and detect bone‑marrow suppression.
Ferritin & Transferrin SaturationBaseline, monthlyAssess iron overload risk.
Blood PressureBaseline, every visitDetect hypertension.
Liver Function TestsBaseline, q3 weeksMonitor hepatotoxicity.
Reticulocyte countEvery visitGauge erythropoietic response.
Injections site examEvery visitIdentify local reactions.
Contraception status (women of childbearing potential)Baseline, q3 weeksDue to limited fetal safety data.

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Clinical Pearls

  • Efficacy Horizon: Response is typically seen after 4–6 treatment cycles; optimize follow‑up by documenting a 1.5–2 g/dL Hb increase or ≥2-unit reduction in transfusion requirement.
  • Weight‑Based Dosing: Adhering to a 3 mg/kg schedule rather than a flat dose enhances response in heavier patients and reduces overtreatment in lighter ones.
  • Iron Management: Begin or intensify iron‑chelation therapy when ferritin >1,200 ng/mL or if FERRITIN/P. Accept that iron overload can mask efficacy data; keep ferritin 30 mL/min), but caution in severe CKD–consider close monitoring for potential delayed clearance of metabolites.
  • Injection Etiquette: Using a 27‑gauge needle, avoid rapid injection to prevent post‑injection migratory pain; hold the site for 30 s.
  • Pregnancy & Lactation: Insufficient human data; counsel patients on no-oral‑concurrency; otherwise, benefit‑risk must be thoroughly discussed.
  • Switching Strategy: For patients failing first‑line erythropoiesis‑stimulating agents, a switch to Yupelri can be considered if they possess a high transfusion burden and no contraindications.

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Key Takeaway

Yupelri offers a unique, ligand‐trap strategy to restore erythropoiesis by dampening the TGF‑β/SMAD 2/3 over‑inhibition loop. Its subcutaneous, weight‑based regimen, coupled with robust monitoring of hematologic and iron parameters, makes it a powerful tool in managing transfusion‑dependent anemia in RiMDS and β‑thalassemia.

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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