Sulfamethoxazole and trimethoprim

Sulfamethoxazole

Generic Name

Sulfamethoxazole

Mechanism

  • Trimethoprim inhibits bacterial dihydrofolate reductase (DHFR), blocking the conversion of dihydrofolate → tetrahydrofolate.
  • Sulfamethoxazole competes with para‑aminobenzoic acid (PABA) for dihydropteroate synthase (DHPS), preventing dihydropteroate formation, the precursor to folic acid.
  • By antagonizing two sequential steps in folate biosynthesis, SMX/TMP causes a potent antimicrobial effect even at low concentrations.

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Pharmacokinetics

ParameterTypical Values (adult)
Absorption>90 % oral; peak plasma 1–2 h
DistributionVolume of distribution: SMX ≈ 2.4 L/kg, TMP ≈ 1.4 L/kg
Protein BindingSMX ~ 10 %; TMP ~ 20 %
MetabolismHepatic 2‑hydroxylation (SMX) and 2‑hydroxy‑SMX; TMP largely unchanged
Renal Excretion70‑80 % unchanged; SMX metabolite 20‑30 %
Half‑lifeSMX ~5–7 h; TMP ~8–9 h (shorter in healthy adults)
Special PopulationsContrast‑enhanced CT may increase SMX half‑life; renal impairment → dose reduction

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Indications

  • Urinary Tract Infection (UTI) (cystitis & pyelonephritis)
  • Community‑acquired Pneumonia (CAP)
  • Sinusitis & Otitis Media (where sensitivity data support use)
  • Clostridioides difficile colitis (especially in combination with metronidazole or vancomycin)
  • Pneumocystis jirovecii prophylaxis in HIV/AIDS, transplant recipients, and patients on high‑dose steroids or immunomodulators.
  • Leishmaniasis (in combination with paromomycin).

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Contraindications

CategoryGuideline
Allergy to sulfonamidesSMX/TMP contraindicated in classic sulfa hypersensitivity (rash, eosinophilia, hemolysis).
G6PD deficiencyTMP induces oxidative stress → hemolytic anemia; avoid or carefully monitor.
PregnancyCategory B (animal data) but avoid in 1st trimester if possible; high‑dose TMP can cause neonatal hyperbilirubinemia.
HypertensionTMP may exacerbate resistant hypertension; avoid with other potassium‑sparing agents.
Renal or hepatic impairmentDose adjustment required; avoid in severe CKD (eGFR < 30 mL/min without dose reduction).

Warnings:
Drug interactions: Sulfonamides potentiate effects of NSAIDs, diuretics, warfarin, and antiretrovirals.
Photosensitivity: Protect from sun exposure.
B12 deficiency: Chronic use can impair absorption; monitor in long‑term prophylaxis.

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Dosing

IndicationStandard DoseFrequencyNotes
UTI (cystitis)1 g SMX + 5 g TMP twice dailyBIDUse 1 : 5 ratio.
UTI (pyelonephritis)2 g SMX + 10 g TMP twice dailyBID7‑10 days.
CAP, sinusitis, otitis media1 g SMX + 5 g TMP twice dailyBID, 7–10 daysAdjust by renal function.
C. difficile1 g SMX + 5 g TMP, 5 am/5 pm for 6‑10 days2×/dayAlternates with metronidazole/vancomycin.
P. jirovecii prophylaxis80 mg TMP + 400 mg SMX once dailyQDLow‑dose prophylaxis (1 g:5 g).
Renal impairmentReduce dose by 50 % in moderate CKD; monitor closely.Adj.Tailor to eGFR.

Administration:
• Oral tablets or suspension.
• Can be given with or without food; avoids GI upset.

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Adverse Effects

ClassCommonSerious
DermatologicMild rash, itching, photosensitivity.Stevens‑Johnson syndrome, toxic epidermal necrolysis, severe drug‑induced hypersensitivity.
HematologicMild anemia, neutropenia.Agranulocytosis, eosinophilic pneumonia, hemolytic anemia (G6PD).
RenalUrinary frequency, mild pain.Acute interstitial nephritis, crystalluria, oxalate nephropathy.
MetabolicNausea, vomiting, diarrhea, abdominal pain.Hyperkalemia (especially with other K‑retaining drugs).
NeurologicDizziness.Rare seizures (dose‑related).
OtherYeast infections (oral & genital), mouth ulcers, B12 deficiency.

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Monitoring

* Renal function: eGFR before and every 2‑3 weeks (if >30 days therapy).
* Serum electrolytes: Na⁺, K⁺, Cl⁻ (baseline, 1‑2 weeks, then monthly).
* Complete Blood Count (CBC): baseline, 1‑2 weeks after start, then periodically.
* Signs of hypersensitivity: rash, fever, eosinophilia.
* B12 levels: particularly in patients requiring long‑term prophylaxis.

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Clinical Pearls

1. Potassium Trap – TMP is a competitive inhibitor of renal tubular Na⁺/K⁺‑ATPase → hyperkalemia. Keep a close eye on patients on ACE inhibitors, ARBs, spironolactone, or NSAIDs.

2. G6PD Deficieny Check – Never give SMX/TMP to an individual with known G6PD deficiency; if unknown, ask about history of hemolytic anemia in response to antimalarials or sulfa drugs.

3. UTI Algorithm – For uncomplicated cystitis in women and uncomplicated pyelonephritis in men, SMX/TMP remains a first‑line choice, but always confirm susceptibility or use local antibiogram if available.

4. Trimethoprim‑Like Effects – Once daily low‑dose prophylaxis for PCP is less toxic than the 2×/day higher dose recommended for treatment, yet still effective if the patient remains adherent.

5. Allergy Cross‑React – Classic sulfa allergy (rash, Stevens‑Johnson) contraindicates SMX/TMP. A mild, non‑severe sulfa allergy (stomach upset or mild rash) may be tolerated with caution.

6. Photography – Even modest sunlight exposure can trigger photosensitivity; patients should apply broad‑spectrum sunscreen during therapy.

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Key take‑away: SMX/TMP’s dual blockade of folate biosynthesis gives it a broad antibacterial spectrum. Proper dosing based on renal function, vigilant monitoring for hyperkalemia and hypersensitivity, and awareness of contraindications make it a safe, high‑yield therapeutic option for many infections.

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Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

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