Alprazolam

Alprazolam

Generic Name

Alprazolam

Mechanism

Alprazolam is a benzodiazepine that potentiates inhibitory neurotransmission.
• Enhances the activity of the γ‑aminobutyric acid (GABA)‑A receptor complex.
• Increases chloride ion influx, hyperpolarizing neuronal membranes.
• Produces anxiolytic, sedative‑hypnotic, anticonvulsant, and muscle‑relaxant effects.

Pharmacokinetics

  • Absorption – Rapid, peak plasma levels within 1–2 h; bioavailability ≈ 96 %.
  • Distribution – Highly protein‑bound (~ 94 %); crosses blood–brain barrier and placenta.
  • Metabolism – Primarily CYP3A4‑mediated hydroxylation → inactive 3‑hydroxy derivatives.
  • Excretion – Renal (≈ 20 %) and biliary routes; half‑life 11–16 h (range 6–27 h).
  • Drug interactions – CYP3A4 inhibitors ↑ levels; CYP3A4 inducers ↓ levels; potentiate CNS depressants.

Indications

  • Generalized anxiety disorder (short‑term or as-needed).
  • Panic disorder (maintenance and acute relief).
  • Anxiogenic states precipitated by antidepressants or heavy alcohol withdrawal.
  • Adjunct to tertiary pain management in certain postoperative cases (off‑label).

Contraindications

  • Absolute contraindications: Acute narrow‑angle glaucoma, severe hepatic impairment, Concomitant use of mono‑amine oxidase inhibitors (within 14 days).
  • Warnings:
  • Risk of dependence, tolerance, and withdrawal.
  • Cognitive/psychomotor impairment—avoid driving, operating machinery.
  • Pregnancy (Category D) – potential fetal harm; use only if benefits outweigh risks.
  • Lactation – minimal evidence; avoid with nursing infants.

Dosing

ConditionInitial doseTitrationMax doseFormSpecial Populations
GAD / Panic0.25 mg PO BID↑0.25 mg every 48 h as needed4 mg/dayImmediate‑release tablets (0.25/0.5 mg)Elderly: start 0.25 mg BID; consider slower titration
Acute panic0.5–1 mg POrepeat as needed2 mg PO TIDXR‑tablet 1.5‑2 mgChildren < 12 y: not approved
Off‑label
• pain
0.5–1 mg PO as needed4 mg/day--Renal impairment: monitor; Hepatic impairment: ↓ dose

*Take with food to reduce GI upset.*

Adverse Effects

  • Common: Drowsiness, dizziness, fatigue, impaired cognition, dry mouth, blurred vision.
  • Less common: Paradoxical agitation, irritability, ataxia.
  • Serious: Respiratory depression (especially in overdose or with opioids), withdrawal seizures, severe depression, suicidal ideation, hepatotoxicity (rare).

Monitoring

  • Clinical: Anxiety score (HAM-A, GAD‑7), sedation level, functional capacity.
  • Laboratory: Liver function tests (baseline, every 4–6 wk if chronic use).
  • Safety: Watch for withdrawal symptoms upon abrupt cessation; taper 2–4 weeks.
  • Therapeutic drug monitoring: Not routine but advisable in cases of suspected overdose or drug interactions.

Clinical Pearls

1. Taper first, don't stop abruptly – a 2–4 wk taper reduces withdrawal risk; consider 0.5 mg per week decrement.
2. Avoid combining with opioids or alcohol – synergistic CNS depression can precipitate fatal hypoventilation.
3. Use lowest effective dose for shortest duration – guidelines recommend ≤2 weeks for acute panic; extended use warrants psychotherapy integration.
4. Consider pharmacogenomics – CYP3A4 polymorphisms can dramatically alter clearance; dose adjustments may be needed in poor metabolizers.
5. In pregnancy, use instead of tricyclics – limited data suggests benzodiazepines are preferable if treatment is unavoidable, but still carry risks.

These concise, evidence‑based points provide a quick reference for students and clinicians, while the hierarchical Markdown format supports SEO and readability.

Medical & AI Content Disclaimers
Medical Disclaimer: Medical definitions are provided for educational purposes and should not replace professional medical advice, diagnosis, or treatment.

AI Content Disclaimer: Some definitions may be AI-generated and may contain inaccuracies. Always verify with authoritative medical references.

Scroll to Top